Hepatitis B virus X protein sensitizes primary mouse hepatocytes to ethanol- and TNF-alpha-induced apoptosis by a caspase-3-dependent mechanism.

Cellular & molecular immunology

Volumn 2, Issue 1, 2005, Pages 40-48

  Kim, Won Ho ^a   Hong, Feng ^a   Jaruga, Barbara ^a   Zhang, Zheng Sheng ^a   Fan, Sai Jun ^a   Liang, T Jake ^a   Gao, Bin ^a  

^a NATIONAL INSTITUTE ON ALCOHOL ABUSE AND ALCOHOLISM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALCOHOL; CASP3 PROTEIN, MOUSE; CASPASE; CASPASE 3; HEPATITIS B VIRUS X PROTEIN; REACTIVE OXYGEN METABOLITE; TRANSACTIVATOR PROTEIN; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL; APOPTOSIS; ARTICLE; CELL CULTURE; CELL MEMBRANE PERMEABILITY; CYTOLOGY; DRUG EFFECT; GENETICS; LIVER CELL; MALE; METABOLISM; MITOCHONDRION; MOUSE; TRANSGENIC MOUSE;

ANIMALS; APOPTOSIS; CASPASE 3; CASPASES; CELL MEMBRANE PERMEABILITY; CELLS, CULTURED; ETHANOL; HEPATOCYTES; MALE; MICE; MICE, TRANSGENIC; MITOCHONDRIA; REACTIVE OXYGEN SPECIES; TRANS-ACTIVATORS; TUMOR NECROSIS FACTOR-ALPHA;

EID: 33644976369     PISSN: 16727681     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (0)