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Volumn 20, Issue 1, 2006, Pages 151-154
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Nf1 gene inactivation in acute myeloid leukemia cells confers cytarabine resistance through MAPK and mTOR pathways [9]
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Author keywords
[No Author keywords available]
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Indexed keywords
1,4 DIAMINO 1,4 BIS(2 AMINOPHENYLTHIO) 2,3 DICYANOBUTADIENE;
CYTARABINE;
MAMMALIAN TARGET OF RAPAMYCIN;
MITOGEN ACTIVATED PROTEIN KINASE;
NEUROFIBROMIN;
PROPIDIUM IODIDE;
PROTEIN KINASE;
ACUTE GRANULOCYTIC LEUKEMIA;
ANIMAL CELL;
ANTINEOPLASTIC ACTIVITY;
APOPTOSIS;
CANCER RESISTANCE;
CELL CYCLE S PHASE;
CONTROLLED STUDY;
ENZYME INHIBITION;
GENE INACTIVATION;
LETTER;
LEUKEMIA CELL LINE;
MITOSIS INHIBITION;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
TIME;
WILD TYPE;
ACUTE DISEASE;
ANIMAL;
CELL DEATH;
DRUG EFFECT;
DRUG RESISTANCE;
GENETICS;
HUMAN;
METABOLISM;
MYELOID LEUKEMIA;
SIGNAL TRANSDUCTION;
TUMOR CELL LINE;
ACUTE DISEASE;
ANIMALS;
CELL DEATH;
CELL LINE, TUMOR;
CYTARABINE;
DRUG RESISTANCE, NEOPLASM;
HUMANS;
LEUKEMIA, MYELOID;
MICE;
MITOGEN-ACTIVATED PROTEIN KINASES;
NEUROFIBROMIN 1;
PROTEIN KINASES;
SIGNAL TRANSDUCTION;
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EID: 33644974752
PISSN: 08876924
EISSN: 14765551
Source Type: Journal
DOI: 10.1038/sj.leu.2404033 Document Type: Letter |
Times cited : (21)
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References (8)
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