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Volumn 13, Issue 10, 2005, Pages 914-917

Selective reductions in plasma Aβ 1-42 in healthy elderly subjects during longitudinal follow-up: A preliminary report

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN[1-40]; AMYLOID BETA PROTEIN[1-42]; APOLIPOPROTEIN; AMYLOID BETA PROTEIN; APOLIPOPROTEIN E;

EID: 33644877175     PISSN: 10647481     EISSN: 15457214     Source Type: Journal    
DOI: 10.1097/00019442-200510000-00012     Document Type: Article
Times cited : (82)

References (12)
  • 1
    • 16044373524 scopus 로고    scopus 로고
    • Secreted amyloid β-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease
    • Scheuner D, Eckman C, Jensen M, et al: Secreted amyloid β-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease. Nat Med 1996; 2:864-870
    • (1996) Nat Med , vol.2 , pp. 864-870
    • Scheuner, D.1    Eckman, C.2    Jensen, M.3
  • 2
    • 0035815573 scopus 로고    scopus 로고
    • Elevated plasma amyloid β-peptide 1-42 and onset of dementia in adults with Down syndrome
    • Schupf N, Patel B, Silverman W, et al: Elevated plasma amyloid β-peptide 1-42 and onset of dementia in adults with Down syndrome. Neurosci Lett 2001; 301:199-203
    • (2001) Neurosci Lett , vol.301 , pp. 199-203
    • Schupf, N.1    Patel, B.2    Silverman, W.3
  • 3
    • 0034703979 scopus 로고    scopus 로고
    • Ertekin-Taner N, Graff-Radford N, Younkin LH, et al: Linkage of plasma Aβ 42 to a quantitative locus on Chromosome 10 in late-onset Alzheimer's disease pedigrees. Science 2000; 290:2303-2304
    • Ertekin-Taner N, Graff-Radford N, Younkin LH, et al: Linkage of plasma Aβ 42 to a quantitative locus on Chromosome 10 in late-onset Alzheimer's disease pedigrees. Science 2000; 290:2303-2304
  • 4
    • 0032869023 scopus 로고    scopus 로고
    • Plasma amyloid β-peptide 1-42 and incipient Alzheimer's disease
    • Mayeux R, Tang MX, Jacobs DM, et al: Plasma amyloid β-peptide 1-42 and incipient Alzheimer's disease. Ann Neurol 1999; 46:412-416
    • (1999) Ann Neurol , vol.46 , pp. 412-416
    • Mayeux, R.1    Tang, M.X.2    Jacobs, D.M.3
  • 5
    • 0242412140 scopus 로고    scopus 로고
    • Plasma Aβ40 and Aβ42 and Alzheimer's disease: Relation to age, mortality, and risk
    • Mayeux R, Honig LS, Tang MX, et al: Plasma Aβ40 and Aβ42 and Alzheimer's disease: relation to age, mortality, and risk. Neurology 2003; 61:1185-1190
    • (2003) Neurology , vol.61 , pp. 1185-1190
    • Mayeux, R.1    Honig, L.S.2    Tang, M.X.3
  • 6
    • 0029100939 scopus 로고
    • Platelets are the primary source of amyloid β-peptide in human blood
    • Chen M, Inestrosa NC, Ross GS, et al: Platelets are the primary source of amyloid β-peptide in human blood. Biochem Biophys Res Commun 1995; 213:96-103
    • (1995) Biochem Biophys Res Commun , vol.213 , pp. 96-103
    • Chen, M.1    Inestrosa, N.C.2    Ross, G.S.3
  • 7
    • 0033975837 scopus 로고    scopus 로고
    • Plasma and cerebrospinal fluid levels of amyloid β proteins 1-40 and 1-42 in Alzheimer disease
    • Mehta PD, Pirttila T, Mehta SP, et al: Plasma and cerebrospinal fluid levels of amyloid β proteins 1-40 and 1-42 in Alzheimer disease. Arch Neurol 2000; 57:100-105
    • (2000) Arch Neurol , vol.57 , pp. 100-105
    • Mehta, P.D.1    Pirttila, T.2    Mehta, S.P.3
  • 8
    • 12244283089 scopus 로고    scopus 로고
    • Cerebrospinal fluid β-amyloid 42 is reduced before the onset of sporadic dementia: A population-based study in 85-year-olds
    • Skoog I, Davidsson P, Aevarsson O, et al: Cerebrospinal fluid β-amyloid 42 is reduced before the onset of sporadic dementia: a population-based study in 85-year-olds. Dement Geriatr Cogn Disord 2003; 15:169-176
    • (2003) Dement Geriatr Cogn Disord , vol.15 , pp. 169-176
    • Skoog, I.1    Davidsson, P.2    Aevarsson, O.3
  • 9
    • 0035907123 scopus 로고    scopus 로고
    • Amyloid β protein 1-40 and 1-42 levels in matched cerebrospinal fluid and plasma from patients with Alzheimer disease
    • Mehta PD, Pirttila T, Patrick BA, et al: Amyloid β protein 1-40 and 1-42 levels in matched cerebrospinal fluid and plasma from patients with Alzheimer disease. Neurosci Lett 2001; 304:102-106
    • (2001) Neurosci Lett , vol.304 , pp. 102-106
    • Mehta, P.D.1    Pirttila, T.2    Patrick, B.A.3
  • 10
    • 4444276735 scopus 로고    scopus 로고
    • Clearance of Alzheimer's Aβ peptide: The many roads to perdition
    • Tanzi RE, Moir RD, Wagner SL: Clearance of Alzheimer's Aβ peptide: the many roads to perdition. Neuron 2004; 43:605-608
    • (2004) Neuron , vol.43 , pp. 605-608
    • Tanzi, R.E.1    Moir, R.D.2    Wagner, S.L.3
  • 11
    • 0347600788 scopus 로고    scopus 로고
    • Potential role of endogenous and exogenous Aβ binding molecules in Aβ clearance and metabolism
    • Edited by Saido T. Georgetown, Tex, Landes Bioscience
    • DeMattos DB, Bales KR, Paul SM, et al: Potential role of endogenous and exogenous Aβ binding molecules in Aβ clearance and metabolism, in Metabolic Regulation of Amyloid Protein and Alzheimer's Disease. Edited by Saido T. Georgetown, Tex, Landes Bioscience, 2003, pp 127-143
    • (2003) Metabolic Regulation of Amyloid Protein and Alzheimer's Disease , pp. 127-143
    • DeMattos, D.B.1    Bales, K.R.2    Paul, S.M.3
  • 12
    • 0035863055 scopus 로고    scopus 로고
    • Age-dependent changes in brain, CSF, and plasma amyloi(beta) protein in the Tg2576 transgenic mouse model of Alzheimer's disease
    • Kawarabayashi T, Younkin LH, Saido TC, Shoji M, Ashe KH, Younkin SG: Age-dependent changes in brain, CSF, and plasma amyloi(beta) protein in the Tg2576 transgenic mouse model of Alzheimer's disease. J Neurosci 2001; 21:372-381
    • (2001) J Neurosci , vol.21 , pp. 372-381
    • Kawarabayashi, T.1    Younkin, L.H.2    Saido, T.C.3    Shoji, M.4    Ashe, K.H.5    Younkin, S.G.6


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.