Apoptosis: a possible mechanism of suppressing parathyroid hyperplasia by calcimimetics

Clinical calcium

Volumn 15 Suppl 1, Issue , 2005, Pages

  Mizobuchi, Masahide ^a   Saji, Fumie ^a   Shiizaki, Kazuhiro ^a   Sato, Toshihiro ^a   Shibata, Maki ^a   Negi, Shigeo ^a   Akizawa, Tadao ^a   Hatamura, Ikuji ^a   Ogata, Hiroaki ^a   Kinugasa, Eriko ^a  

^a WAKAYAMA MEDICAL UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

ANILINE DERIVATIVE; CALCITRIOL; CALCIUM SENSING RECEPTOR; N (2 CHLOROPHENYLPROPYL) 1 (3 METHOXYPHENYL)ETHYLAMINE; N-(2-CHLOROPHENYLPROPYL)-1-(3-METHOXYPHENYL)ETHYLAMINE; PARATHYROID HORMONE;

ANIMAL; APOPTOSIS; ARTICLE; CELL CULTURE; CYTOLOGY; DISEASE MODEL; DOSE RESPONSE; DRUG EFFECT; DRUG POTENTIATION; MALE; METABOLISM; PARATHYROID GLAND; PATHOLOGY; RAT; SECONDARY HYPERPARATHYROIDISM; SECRETION; SPRAGUE DAWLEY RAT;

ANILINE COMPOUNDS; ANIMALS; APOPTOSIS; CALCITRIOL; CELLS, CULTURED; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; HYPERPARATHYROIDISM, SECONDARY; MALE; PARATHYROID GLANDS; PARATHYROID HORMONE; RATS; RATS, SPRAGUE-DAWLEY; RECEPTORS, CALCIUM-SENSING;

EID: 33644814449     PISSN: 09175857     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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