Diastolic dysfunction and abnormality of the Na+/Ca2+ exchanger in single uremic cardiac myocytes

Kidney International

Volumn 69, Issue 5, 2006, Pages 846-851

  McMahon, A C ^a,c   Naqvi, R U ^b   Hurst, M J ^a   Raine, A E G ^a   MacLeod, K T ^b  

^a QUEEN MARY UNIVERSITY OF LONDON   (United Kingdom)

^b NATIONAL HEART AND LUNG INSTITUTE   (United Kingdom)

^c Department of Microbiology and Infectious Diseases   (Australia)

Author keywords

Calcium; Hypertrophy; Myocyte; Na+ Ca2+ exchange; Uremia

Indexed keywords

INDO 1; SODIUM CALCIUM EXCHANGE PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CALCIUM TRANSPORT; CELL STRUCTURE; CONTROLLED STUDY; DIASTOLE; FUNCTIONAL PROTEOMICS; HEART DISEASE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART MUSCLE CONTRACTURE; HEART MUSCLE RELAXATION; HEART PACING; HEART VENTRICLE; HYPERTROPHY; MALE; NEPHRECTOMY; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; RAT; UREMIA;

ANIMALS; CALCIUM SIGNALING; CARDIOVASCULAR DISEASES; CELL ENLARGEMENT; DIASTOLE; HUMANS; ION TRANSPORT; KINETICS; MALE; MYOCYTES, CARDIAC; RATS; RATS, WISTAR; SODIUM-CALCIUM EXCHANGER; UREMIA; VASODILATION;

EID: 33644560686     PISSN: 00852538     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.ki.5000193     Document Type: Article

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