Constitutive activation of rac1 results in mitochondrial oxidative stress and induces premature endothelial cell senescence.

Arteriosclerosis, thrombosis, and vascular biology

Volumn 23, Issue 1, 2003, Pages

  Deshpande, Shailesh S ^a   Qi, Bing ^a   Park, Young Chul ^a   Irani, Kaikobad ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

RAC1 PROTEIN; REACTIVE OXYGEN METABOLITE;

ARTICLE; CELL AGING; CELL LINE; CHEMISTRY; HUMAN; METABOLISM; MITOCHONDRION; OXIDATIVE STRESS; PATHOLOGY; PHYSIOLOGY; PREMATURE AGING; UMBILICAL VEIN; VASCULAR ENDOTHELIUM;

AGING, PREMATURE; CELL AGING; CELL LINE; ENDOTHELIUM, VASCULAR; HUMANS; MITOCHONDRIA; OXIDATIVE STRESS; RAC1 GTP-BINDING PROTEIN; REACTIVE OXYGEN SPECIES; UMBILICAL VEINS;

MLCS; MLOWN;

EID: 3142557059     PISSN: None     EISSN: 15244636     Source Type: Journal    
DOI: 10.1161/01.atv.0000047869.13737.53     Document Type: Article

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