Reduction of Caenorhabditis elegans frataxin increases sensitivity to oxidative stress, reduces lifespan, and causes lethality in a mitochondrial complex II mutant

FASEB Journal

Volumn 20, Issue 1, 2006, Pages 172-174

  Vázquez Manrique, Rafael P ^a,b   González Cabo, Pilar ^a   Ros, Sheila ^a   Aziz, Homera ^b   Baylis, Howard A ^b   Palau, Francesc ^a  

^a INSTITUTO DE BIOMEDICINA DE VALENCIA   (Spain)

^b UNIVERSITY OF CAMBRIDGE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

FRATAXIN; HYBRID PROTEIN; PARAQUAT;

ANIMAL EXPERIMENT; ARTICLE; CAENORHABDITIS ELEGANS; DEFECATION; ELECTRON TRANSPORT; FRIEDREICH ATAXIA; LETHALITY; LIFESPAN; NONHUMAN; NUCLEOTIDE SEQUENCE; OXIDATIVE STRESS; PHENOTYPE; PLEIOTROPY; PRIORITY JOURNAL; PROTEIN DEFICIENCY; PROTEIN EXPRESSION; PROTEIN FUNCTION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; RNA INTERFERENCE;

ANIMALS; CAENORHABDITIS ELEGANS; CAENORHABDITIS ELEGANS PROTEINS; GENE EXPRESSION REGULATION, ENZYMOLOGIC; GENES, LETHAL; GENOTYPE; IRON-BINDING PROTEINS; LONGEVITY; MITOCHONDRIA; MUTATION; ORGANISMS, GENETICALLY MODIFIED; OXIDATIVE STRESS; PHENOTYPE; RNA INTERFERENCE; SUCCINATE DEHYDROGENASE;

ANIMALIA; ATAXIA; CAENORHABDITIS ELEGANS;

EID: 30744458144     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.05-4212fje     Document Type: Article

References (0)