Circulating mononuclear cells with a dual, macrophage-fibroblast phenotype contribute robustly to hypoxia-induced pulmonary adventitial remodeling

Chest

Volumn 128, Issue 6 SUPPL., 2005, Pages 583S-584S

  Frid, Maria G ^a   Brunetti, Jacqueline A ^a   Burke, Danielle L ^a   Carpenter, Todd C ^a   Davie, Neil J ^a   Stenmark, Kurt R ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL CELL; CONFERENCE PAPER; CONFOCAL LASER MICROSCOPY; CONTROLLED STUDY; COR PULMONALE; FIBROBLAST; FIBROMUSCULAR DYSPLASIA; IMMUNOHISTOCHEMISTRY; LUNG VASCULAR RESISTANCE; MACROPHAGE; MONONUCLEAR CELL; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PULMONARY ARTERY; PULMONARY HYPERTENSION;

EID: 30144442219     PISSN: 00123692     EISSN: None     Source Type: Journal    
DOI: 10.1378/chest.128.6_suppl.583S     Document Type: Conference Paper

References (3)

1. Quan TE, Cowper S, Wu S-P, et al. Circulating fibrocytes: collagen-secreting cells of the peripheral blood. J Biochem Cell Biol 2004; 36:598-606
2. Hashimoto N, Jin H, Liu T, et al. Bone marrow-derived progenitor cells in pulmonary fibrosis. J Clin Invest 2004; 113:243-252
3. Davie NJ, Crossno JT, Frid MG, et al. Hypoxia-induced pulmonary artery adventitial remodeling and neovascularization: contribution of progenitor cells. Am J Physiol Lung Cell Mol Physiol 2004; 286;L668-L678