Microtubule disarray in primary cultures of human hepatocytes inhibits transcriptional activity of the glucocorticoid receptor via activation of c-jun N-terminal kinase.

Biomedical papers of the Medical Faculty of the University Palacký, Olomouc, Czechoslovakia

Volumn 148, Issue 2, 2004, Pages 135-139

  Dvorák, Zdenek ^a   Maurel, Patrick ^a   Ulrichová, Jitka ^a   Modrianský, Martin ^a  

^a PALACKÝ UNIVERSITY   (Czech Republic)

Author keywords

[No Author keywords available]

Indexed keywords

ANTHRA(1,9 CD)PYRAZOL 6(2H) ONE; ANTHRA(1,9-CD)PYRAZOL-6(2H)-ONE; ANTHRACENE DERIVATIVE; COLCHICINE; GLUCOCORTICOID RECEPTOR; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; STRESS ACTIVATED PROTEIN KINASE; TYROSINE AMINOTRANSFERASE;

ARTICLE; CELL CULTURE; DRUG ANTAGONISM; DRUG EFFECT; ENZYME ACTIVATION; HUMAN; LIVER CELL; METABOLISM; MICROTUBULE; PHYSIOLOGY; TRANSACTIVATION; ULTRASTRUCTURE;

ANTHRACENES; CELLS, CULTURED; COLCHICINE; ENZYME ACTIVATION; HEPATOCYTES; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; MICROTUBULES; NF-KAPPA B; RECEPTORS, GLUCOCORTICOID; TRANS-ACTIVATION (GENETICS); TYROSINE TRANSAMINASE;

EID: 27944465892     PISSN: 12138118     EISSN: None     Source Type: Journal    
DOI: 10.5507/bp.2004.024     Document Type: Article

References (0)