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Volumn 19, Issue 13, 2005, Pages 1908-1910
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NFiκB and AP-1 differentially contribute to the induction of Mn-SOD and eNOS during the development of oxidant tolerance
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NONE
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Author keywords
[No Author keywords available]
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Indexed keywords
ENDOTHELIAL NITRIC OXIDE SYNTHASE;
HYDROGEN PEROXIDE;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
MANGANESE SUPEROXIDE DISMUTASE;
TRANSCRIPTION FACTOR AP 1;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
OXIDIZING AGENT;
SUPEROXIDE DISMUTASE;
ANOXIA;
ARTICLE;
CONTROLLED STUDY;
ENZYME ACTIVITY;
HEART MUSCLE CELL;
INFLAMMATION;
OXIDATIVE STRESS;
PHENOTYPE;
PRIORITY JOURNAL;
REOXYGENATION;
SIGNAL TRANSDUCTION;
ACTIVE TRANSPORT;
ANIMAL;
BIOLOGICAL MODEL;
C57BL MOUSE;
CELL NUCLEUS;
CHEMISTRY;
DOSE RESPONSE;
METABOLISM;
MOUSE;
NEWBORN;
PROTEIN TRANSPORT;
STATISTICAL MODEL;
TIME;
ACTIVE TRANSPORT, CELL NUCLEUS;
ANIMALS;
ANIMALS, NEWBORN;
CELL NUCLEUS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
INFLAMMATION;
MICE;
MICE, INBRED C57BL;
MODELS, BIOLOGICAL;
MODELS, STATISTICAL;
MYOCYTES, CARDIAC;
NF-KAPPA B;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
NITRIC OXIDE SYNTHASE TYPE III;
OXIDANTS;
OXIDATIVE STRESS;
PHENOTYPE;
PROTEIN TRANSPORT;
SUPEROXIDE DISMUTASE;
TIME FACTORS;
TRANSCRIPTION FACTOR AP-1;
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EID: 27744591589
PISSN: 08926638
EISSN: None
Source Type: Journal
DOI: 10.1096/fj.05-4028fje Document Type: Article |
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Times cited : (26)
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References (0)
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