Lack of association of TPH2 exon XI polymorphisms with major depression and treatment resistance [3]

Molecular Psychiatry

Volumn 10, Issue 11, 2005, Pages 976-977

  Garriock, H A ^a   Allen, J J B ^a   Delgado, P ^a   Nahaz, Z ^a   Kling, M A ^a   Carpenter, L ^a   Burke, M ^a   Burke, W ^a   Schwartz, T ^a   Marangell, L B ^a   Husain, M ^a   Erickson, R P ^a   Moreno, F A ^a  

^a UNIVERSITY OF ARIZONA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIDEPRESSANT AGENT; SEROTONIN; SEROTONIN UPTAKE INHIBITOR; TRYPTOPHAN HYDROXYLASE; TRYPTOPHAN HYDROXYLASE 2; UNCLASSIFIED DRUG;

CONTROLLED STUDY; ETHNIC DIFFERENCE; EXON; FEMALE; GENE FREQUENCY; GENE LOCUS; GENE SEQUENCE; GENETIC POLYMORPHISM; GENOTYPE; HUMAN; LETTER; MAJOR CLINICAL STUDY; MAJOR DEPRESSION; MALE; PRIORITY JOURNAL; SEX RATIO; SINGLE NUCLEOTIDE POLYMORPHISM;

ALLELES; ANTIDEPRESSIVE AGENTS, SECOND-GENERATION; BIPOLAR DISORDER; CASE-CONTROL STUDIES; DEPRESSIVE DISORDER, MAJOR; DRUG RESISTANCE; EXONS; FEMALE; GENE FREQUENCY; HUMANS; MALE; POLYMORPHISM, SINGLE NUCLEOTIDE; SEROTONIN UPTAKE INHIBITORS; TRYPTOPHAN HYDROXYLASE;

EID: 27544495122     PISSN: 13594184     EISSN: 14765578     Source Type: Journal    
DOI: 10.1038/sj.mp.4001712     Document Type: Letter

References (3)

1. Zhang X et al. Neuron 2005; 45: 11-16.
2. Zhang X et al. Science 2004; 305: 217.
3. http://www.ncbi.nlm.nih.gov/projects/SNP/.