Ischemic optic neuropathy: "Whodunit?";Neuropathie optique ischémique: Qui est coupable?

Canadian Journal of Anesthesia

Volumn 51, Issue 4, 2004, Pages 286-289

  Tempelhoff, René ^a  

^a BARNES JEWISH HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CARBON DIOXIDE;

ANESTHESIA INDUCTION; ARTERIAL CARBON DIOXIDE TENSION; ARTERY BLOOD FLOW; ARTERY DIAMETER; ARTERY STEAL SYNDROME; BLINDNESS; BLOOD FLOW VELOCITY; BLOOD PRESSURE MONITORING; BRACHIAL ARTERY; BRAIN BLOOD FLOW; BRAIN BLOOD VESSEL; EDITORIAL; EXTERNAL CAROTID ARTERY; HISTOPATHOLOGY; HUMAN; HYPERCAPNIA; HYPOCAPNIA; INCIDENCE; INTRAOCULAR PRESSURE ABNORMALITY; ISCHEMIC OPTIC NEUROPATHY; OPHTHALMIC ARTERY; OPTIC NERVE INJURY; PRIORITY JOURNAL; VASOCONSTRICTION;

EID: 2542636356     PISSN: 0832610X     EISSN: None     Source Type: Journal    
DOI: 10.1007/BF03018231     Document Type: Editorial

References (3)

1. Lee AL, Vavilata MS, Lam AM, et al. Ophthalmic artery blood flow velocity increases during hypocapnia. J Can Anesth 2004; 51: 388-92.
2. Cheng MA, Todorov A, Tempelhoff R, McHugh T, Crowder CM, Lauryssen C. The effect of prone positioning on intraocular pressure in anesthetized patients. Anesthesiology 2001; 95: 1351-5.
3. Akca O, Doufas AG, Morioka N, Iscoe S, Fisher J, Sessler DI. Hypercapnia improves tissue oxygenation. Anesthesiology 2002; 97: 801-6.