Staphylococcus aureus and lipopolysaccharide induce homologous tolerance but heterologous priming: role of interferon-gamma.

Shock (Augusta, Ga.)

Volumn 21, Issue 3, 2004, Pages 254-260

  Peck, Octavia M ^a   Fan, Hongkuan ^a   Tempel, George E ^a   Teti, Giuseppe ^a   Halushka, Perry V ^a   Cook, James A ^a  

^a MEDICAL UNIVERSITY OF SOUTH CAROLINA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

GAMMA INTERFERON; LIPOPOLYSACCHARIDE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE KINASE; MITOGEN ACTIVATED PROTEIN KINASE KINASE 4; MITOGEN ACTIVATED PROTEIN KINASE P38; STRESS ACTIVATED PROTEIN KINASE; THROMBOXANE B2; TUMOR NECROSIS FACTOR ALPHA;

ARTICLE; CELL LINE; HUMAN; MACROPHAGE; METABOLISM; MICROBIOLOGY; PHOSPHORYLATION; SIGNAL TRANSDUCTION; STAPHYLOCOCCUS AUREUS; WESTERN BLOTTING;

BLOTTING, WESTERN; CELL LINE; HUMANS; INTERFERON TYPE II; JNK MITOGEN-ACTIVATED PROTEIN KINASES; LIPOPOLYSACCHARIDES; MACROPHAGES; MAP KINASE KINASE 4; MAP KINASE SIGNALING SYSTEM; MITOGEN-ACTIVATED PROTEIN KINASE KINASES; MITOGEN-ACTIVATED PROTEIN KINASES; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; SIGNAL TRANSDUCTION; STAPHYLOCOCCUS AUREUS; THROMBOXANE B2; TUMOR NECROSIS FACTOR-ALPHA;

EID: 2442442903     PISSN: 10732322     EISSN: None     Source Type: Journal    
DOI: 10.1097/01.shk.0000111662.09279.59     Document Type: Article

References (0)