The effect of insulin and β-adrenergic stimulation on glucose transport, glut4 and PKB activation in the myocardium of lean and obese Non-insulin dependent diabetes mellilus rat
Huisman B, Zyl M V, Keyser A, et al. The effect of insulin and β-adrenergic stimulation on glucose transport, glut4 and PKB activation in the myocardium of lean and obese Non-insulin dependent diabetes mellilus rat. Mol Cell Biochem, 2001,223:15.
Comparison of glucose transporter containing vesicles from rat fat and muscle tissues evidence for a unique endosomal compartment
Kandror K V, Coderre L, Pushkin A V, et al. Comparison of glucose transporter containing vesicles from rat fat and muscle tissues evidence for a unique endosomal compartment. Biochem J, 1995,307:383.
Insulin action on glucose transport and plasma membrane GLUT4 content in skeletal muscle from patients with NIDDM
Zierath J R, He L, Gumà A, et al. Insulin action on glucose transport and plasma membrane GLUT4 content in skeletal muscle from patients with NIDDM. Diabetologia, 1996,39:1180.
Defective insulin-induced GLUT4 translocation in skeletal muscle of high fat-fed rats is associated with alterations in both Akt/protein kinase B and atypical protein kinase C (ζ/λ) activities
Tremblay F, Lavigne C, Jacques H, et al. Defective insulin-induced GLUT4 translocation in skeletal muscle of high fat-fed rats is associated with alterations in both Akt/protein kinase B and atypical protein kinase C (ζ/λ) activities. Diabetes, 2001, 50:1901.
Sustained exposure of L6 myotubes to high glucose and insulin decreases insulin-stimulated GLUT4 translocation but upregulates GLUT4 activity
Huang C, Somwar R, Patel N, et al. Sustained exposure of L6 myotubes to high glucose and insulin decreases insulin-stimulated GLUT4 translocation but upregulates GLUT4 activity. Diabetes, 2002, 51 (7): 2090.