Alternative splicing, a new pharmacological target for small chemical molecules;L'épissage alternatif, une nouvelle cible pharmacologique pour des petites molécules chimiques

Bulletin du Cancer

Volumn 92, Issue 7-8, 2005, Pages 631-

  Jeanteur, Philippe ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

ANTINEOPLASTIC AGENT; DNA TOPOISOMERASE;

ALTERNATIVE RNA SPLICING; CANCER CHEMOTHERAPY; DRUG TARGETING; FLUORESCENCE SPECTROSCOPY; GENETIC TRANSCRIPTION; IN VIVO STUDY; INTRON; NOTE; PATHOLOGY; PROTEIN PHOSPHORYLATION;

EID: 24144434721     PISSN: 00074551     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Note

References (5)

1. Rossi, F. et al. Mammalian DNA topoisomerase I specifically phosphorylates SR protein splicing factors. Nature 1996; 381: 80-2.
2. Jeanteur P. L'ADN topoisomérase I: une cible double pout les drogues anti-cancéreuses. Bull Cancer 1997; 84: 190.
3. Soret J et al. Altered protein phosphorylation and exonic enhancer-dependent splicing in mammalian cells lacking topoisomerase I. Cancer Res 200363: 8203-11.
4. Jeanteur P. La topoisomérase I au cœur de l'épissage alternatif. Bull Cancer 2003; 90: 1033.
5. Soret J et al. Selective modification of alternative splicing by indole derivatives that target serine-arginine-rich protein splicing factors. Proc Natl Acad Sci USA 2005; 102: 8764-69.