Protective effects of salidroside on injury induced by hypoxia/hypoglycemia in cultured neurons

Zhongguo Zhongyao Zazhi

Volumn 29, Issue 5, 2004, Pages 461-462

  Zhang, Wen Sheng ^a   Zhu, Ling Qun ^b   Niu, Fu Ling ^b   Deng, Rui Chun ^a   Ma, Chuan Xiang ^a  

^a BEIJING NORMAL UNIVERSITY   (China)

^b BEIJING UNIVERSITY OF CHINESE MEDICINE   (China)

Author keywords

Apoptosis; Calcium; Hypoxia hypoglycemia; Salidroside; SH SY5Y

Indexed keywords

CALCIUM; GLUCOSIDE; LACTATE DEHYDROGENASE; NEUROPROTECTIVE AGENT; PHENOL DERIVATIVE; RHODIOLOSIDE;

APOPTOSIS; ARTICLE; CELL HYPOXIA; CHEMISTRY; DRUG EFFECT; HUMAN; HYPOGLYCEMIA; ISOLATION AND PURIFICATION; MEDICINAL PLANT; METABOLISM; NERVE CELL; PATHOLOGY; RHODIOLA;

APOPTOSIS; CALCIUM; CELL HYPOXIA; GLUCOSIDES; HUMANS; HYPOGLYCEMIA; L-LACTATE DEHYDROGENASE; NEURONS; NEUROPROTECTIVE AGENTS; PHENOLS; PLANTS, MEDICINAL; RHODIOLA;

EID: 23844451185     PISSN: 10015302     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (10)

1. Tenneti L,D'Emilia D M, Lipton S A.Suppression of neuronl apoptosis by S-nitroslalion of caspases. Neurosci Lett,1997,236(3):139.
2. 2+ ionophre-induced apoptosis on cultured embryonic rat cortical neurons. Brain Res, 1994,652(1):65.
3. Yavin E, Billia D M. Apoptotic death in cerebral hemisphere cells is density dependent and modulated by transient oxygen and glucose deprivation. J Neurosci Res,1997,47(5):471.