Protective effects of rhynchophylline on cerebral ischemia-reperfusion injury

Chinese Pharmaceutical Journal

Volumn 40, Issue 11, 2005, Pages 833-835

  Wu, Er Bing ^a   Sun, An Sheng ^a   Wu Qin, ^a   Yu, Li Mei ^a   Shi, Jing Shan ^a   Huang, Xie Nan ^a  

^a ZUNYI MEDICAL UNIVERSITY   (China)

Author keywords

Anti oxidative activity; Cerebral ischemia reperfusion; Rhynchophlline

Indexed keywords

BIOLOGICAL ORGANS; BRAIN; OXIDATION; PATHOLOGY;

ANTI-OXIDATIVE ACTIVITY; CEREBRAL ISCHEMIA-REPERFUSION; ISCHEMIA-REPERFUSION INJURY; RHYNCHOPHYLLINE;

DRUG PRODUCTS;

LACTATE DEHYDROGENASE; MALONALDEHYDE; NIMODIPINE; NITRIC OXIDE; RHYNCHOPHYLLINE; SUPEROXIDE DISMUTASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN ISCHEMIA; BRAIN REGION; CAROTID ARTERY OBSTRUCTION; COMMON CAROTID ARTERY; CONTROLLED STUDY; DOSE RESPONSE; DRUG EFFECT; DRUG MECHANISM; ENZYME ACTIVITY; HIPPOCAMPUS; LIPID PEROXIDATION; NEUROPATHOLOGY; NEUROPROTECTION; NONHUMAN; REPERFUSION INJURY; SURVIVAL;

EID: 23644455308     PISSN: 10012494     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (10)

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2. Colbourne F, Sutherland GR, Auer RN. Electron microscopic evidence against apoptosis as the mechanism of neuronal death in global ischemia[J]. J Neuroscience,1999,19(11):4200.
3. Yagita Y, Matsumoto M, Kitagawa K, et al. DNA cleavage and proteolysis of microtubule associated protein 2 after cerebral ischemia of different severity[J]. Neuroscience,1999,92(4):1417.