Developmental origins and environmental influences - Introduction

Birth Defects Research Part A - Clinical and Molecular Teratology

Volumn 73, Issue 7, 2005, Pages 469-

  Heindel, Jerrold J ^a   Levin, Edward ^b  

^a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

^b DUKE UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ENDOCRINE DISRUPTOR; ESTROGEN DERIVATIVE;

ADRENERGIC SYSTEM; APPETITE; CELL MATURATION; CONFERENCE PAPER; DEVELOPED COUNTRY; ENVIRONMENTAL EXPOSURE; ENVIRONMENTAL FACTOR; EPIDEMIC; EXERCISE; FOOD INTAKE; GENETIC PREDISPOSITION; GENOTYPE ENVIRONMENT INTERACTION; HUMAN; INCIDENCE; LIPID METABOLISM; NONHUMAN; NUTRITION; OBESITY; PHYSICAL ACTIVITY; PRIORITY JOURNAL; UNITED STATES;

ADIPOCYTES; ENVIRONMENTAL EXPOSURE; GENETIC PREDISPOSITION TO DISEASE; HUMANS; MODELS, ANIMAL; OBESITY; RISK FACTORS;

EID: 22944450014     PISSN: 15420752     EISSN: None     Source Type: Journal    
DOI: 10.1002/bdra.20141     Document Type: Conference Paper

References (3)

1. Baillie-Hamilton PF. 2002. Chemical toxins: a hypothesis to explain the global obesity epidemic. J Altern Complement Med 8:185-192.
2. DHHS. US. Department of Health and Human Services (DHHS). The Surgeon General's call to action to prevent and decrease overweight and obesity. Available at: http://www.surgeongeneral.gov/topics/obesity. Accessed: March 11, 2005.
3. NIH. National Institutes of Health (NIH). NIH obesity research. Strategic plan for NIH obesity research. Available at: http://obesityresearch. nih.gov. Accessed: March 11, 2005.