Host γδ T cells: An innate bridge to the epithelial targets of GVHD?

Blood

Volumn 106, Issue 2, 2005, Pages 393-394

  Hill, Geoffrey R ^a  

^a QIMR BERGHOFER MEDICAL RESEARCH INSTITUTE   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE; CYTOKINE; INTERLEUKIN 10; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; T LYMPHOCYTE RECEPTOR DELTA CHAIN; T LYMPHOCYTE RECEPTOR GAMMA CHAIN;

AMINO ACID SEQUENCE; ANTIGEN EXPRESSION; ANTIGEN PRESENTING CELL; CYTOKINE RELEASE; EPITHELIUM CELL; GRAFT VERSUS HOST REACTION; HOST CELL; HUMAN; IMMUNE RESPONSE; IMMUNITY; INNATE IMMUNITY; INTESTINE; MAJOR HISTOCOMPATIBILITY COMPLEX; MUCOSAL IMMUNITY; NOTE; PRIORITY JOURNAL; SKIN; T LYMPHOCYTE;

EID: 22144455029     PISSN: 00064971     EISSN: None     Source Type: Journal    
DOI: 10.1182/blood-2005-04-1616     Document Type: Note

References (4)

1. Shlomchik WD, Couzens MS, Tang CB, et al. Prevention of graft versus host disease by inactivation of host antigen presenting cells. Science. 1999;285:412-415.
2. Carding SR, Egan PJ. Gammadelta T cells: functional plasticity and heterogeneity. Nat Rev Immunol. 2002;2:336-345.
3. Hayday A, Tigelaar R. Immunoregulation in the tissues by gammadelta T cells. Nat Rev Immunol. 2003;3:233-242.
4. Hill GR, Ferrara JLM. The primacy of the gastrointestinal tract as a target organ of graft-versus-host disease: rationale for the use of cytokine shields in allogeneic bone marrow transplantation. Blood. 2000;95:2754-2759.