Effect of baicalin and dexamethasone on cytokines in brain tissue of infectious brain edema

Bulletin of Hunan Medical University

Volumn 25, Issue 6, 2000, Pages 519-521

  Yu, Y ^a   Yang, Y J ^a   Tao, Y G ^a  

^a CENTRAL SOUTH UNIVERSITY   (China)

Author keywords

Baicalin; Brain edema; Dexamethasone; Interleukin 1 ; Rat; Tumor necrosis factor

Indexed keywords

BAICALIN; CYTOKINE; DEXAMETHASONE; INTERLEUKIN 1BETA; SODIUM ION; TUMOR NECROSIS FACTOR ALPHA; WATER; FLAVONE DERIVATIVE; INTERLEUKIN 1; NEUROPROTECTIVE AGENT; TUMOR NECROSIS FACTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN EDEMA; BRAIN INFECTION; BRAIN WATER; CONTROLLED STUDY; CORTICOSTEROID THERAPY; DRUG EFFICACY; IMMUNOMODULATION; NONHUMAN; PROTEIN EXPRESSION; RAT; ANIMAL; BORDETELLA PERTUSSIS; FEMALE; MALE; METABOLISM; MICROBIOLOGY; PERTUSSIS; RANDOMIZATION; RAT STRAIN;

ANIMAL; BORDETELLA PERTUSSIS; BRAIN EDEMA; DEXAMETHASONE; ENGLISH ABSTRACT; FEMALE; FLAVONES; INTERLEUKIN-1; MALE; NEUROPROTECTIVE AGENTS; RANDOM ALLOCATION; RATS; RATS, SPRAGUE-DAWLEY; SUPPORT, NON-U.S. GOV'T; TUMOR NECROSIS FACTOR; WHOOPING COUGH;

EID: 1842813290     PISSN: 10005625     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (11)

1. The role of nuclear factor-κB in cytokine gene regutation[J]
2. Inflammatory mediators and stroke; new opportunities for novel therapeutics[J]
3. Intracerebral administration of interleukin-1 beta and induction of inflammation, apoptosis, and vasogenic edema[J]
4. Intracarotid tumor necrosis factor-alpha administration increases the blood-brain barrier permeability in cerebral cortex of the newborn pig: Quantitative aspects of double-labeling studies and confocal laser scanning analysis[J]
5. Over expression of interleukin-1 receptor antagonist in the mouse brain reduce ischemic brain injury[J]
6. MK801 and dexamethasone reduce both tumor necrosis factor levels and infarct volume after focal cerebral ischemia in the rat brain[J]
7. Glucocorticoids inhibit the induction of nitric oxide synthase II by down regulating cytokine-induced activity of transcription factor nuclear factor-κB[J]