Mitochondrial function is a critical determinant of IL-1-induced ERK activation

FASEB Journal

Volumn 19, Issue 7, 2005, Pages 837-839

  Wang, Qin ^a   Downey, Gregory P ^b   Bajenova, Elena ^a   Abreu, Maite ^b   Kapus, András ^c   McCulloch, Christopher A ^a  

^a UNIVERSITY OF TORONTO   (Canada)

^b UNIVERSITY HEALTH NETWORK   (Canada)

^c TORONTO GENERAL HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM ION; INTERLEUKIN 1; IONOMYCIN; MATRIX METALLOPROTEINASE; MITOGEN ACTIVATED PROTEIN KINASE; THAPSIGARGIN;

ARTICLE; CALCIUM TRANSPORT; CELL CULTURE; CELL ENERGY; CONTROLLED STUDY; CYTOSOL; DEPOLARIZATION; ENDOPLASMIC RETICULUM; ENZYME ACTIVATION; FIBROBLAST; HUMAN; HUMAN CELL; INFLAMMATION; MITOCHONDRION; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION;

ADENOSINE TRIPHOSPHATE; CALCIUM; CELLS, CULTURED; CHELATING AGENTS; EGTAZIC ACID; ENDOPLASMIC RETICULUM; ENERGY METABOLISM; ENZYME ACTIVATION; EXTRACELLULAR SIGNAL-REGULATED MAP KINASES; FIBROBLASTS; GINGIVA; HUMANS; IMMUNOBLOTTING; INTERLEUKIN-1; KINETICS; MEMBRANE POTENTIALS; MITOCHONDRIA; MITOCHONDRIAL MEMBRANES; SIGNAL TRANSDUCTION;

EID: 18144391514     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.04-2657fje     Document Type: Article

References (0)