Hypoxia-induced renal epithelial cell death through caspase-dependent pathway: role of Bcl-2, Bcl-xL and Bax in tubular injury.

International journal of molecular medicine

Volumn 14, Issue 4, 2004, Pages 633-640

  Yamamoto, Kei ^a   Tomita, Naruya ^a   Yoshimura, Shin ichi ^a   Nakagami, Hironori ^a   Taniyama, Yoshiaki ^a   Yamasaki, Keita ^a   Ogihara, Toshio ^a   Morishita, Ryuichi ^a  

^a OSAKA UNIVERSITY GRADUATE SCHOOL OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BAX PROTEIN, HUMAN; BCL2L1 PROTEIN, HUMAN; CASP3 PROTEIN, HUMAN; CASPASE; CASPASE 3; CYTOCHROME C; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL X;

APOPTOSIS; ARTICLE; CELL HYPOXIA; ENZYMOLOGY; EPITHELIUM CELL; HUMAN; KIDNEY; METABOLISM; PATHOLOGY; PHYSIOLOGY; SIGNAL TRANSDUCTION;

APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; BCL-X PROTEIN; CASPASE 3; CASPASES; CELL HYPOXIA; CYTOCHROMES C; EPITHELIAL CELLS; HUMANS; KIDNEY; PROTO-ONCOGENE PROTEINS C-BCL-2; SIGNAL TRANSDUCTION;

MLCS; MLOWN;

EID: 16644399110     PISSN: 11073756     EISSN: None     Source Type: Journal    
DOI: 10.3892/ijmm.14.4.633     Document Type: Article

References (0)