Wild-type p16INK4a suppresses cell growth, telomerase activity and DNA repair in human breast cancer MCF-7 cells.

International journal of oncology

Volumn 24, Issue 6, 2004, Pages 1597-1605

  Duan, Jianming ^a   Chen, Zhong ^a   Liu, Pinghu ^a   Zhang, Zongyu ^a   Tong, Tanjun ^a  

^a PEKING UNIVERSITY HEALTH SCIENCE CENTER   (China)

Author keywords

[No Author keywords available]

Indexed keywords

DNA BINDING PROTEIN; PROTEIN P16INK4A; RETINOBLASTOMA PROTEIN; TELOMERASE;

ADENOCARCINOMA; AGED; ARTICLE; BREAST TUMOR; CELL ADHESION; CELL CULTURE; CELL CYCLE; DNA REPAIR; DRUG ANTAGONISM; DRUG EFFECT; FEMALE; GENE DELETION; GENE VECTOR; GENETICS; HOMOZYGOTE; HUMAN; METABOLISM; PATHOLOGY; PHOSPHORYLATION; RETROVIRUS; TELOMERE;

ADENOCARCINOMA; AGED; BREAST NEOPLASMS; CELL ADHESION; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P16; DNA REPAIR; DNA-BINDING PROTEINS; FEMALE; GENE DELETION; GENETIC VECTORS; HOMOZYGOTE; HUMANS; PHOSPHORYLATION; RETINOBLASTOMA PROTEIN; RETROVIRIDAE; TELOMERASE; TELOMERE; TUMOR CELLS, CULTURED;

EID: 16544386532     PISSN: 10196439     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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