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Volumn 2, Issue 5, 2003, Pages 465-470
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An intact NF-kappaB pathway is required for histone deacetylase inhibitor-induced G1 arrest and maturation in U937 human myeloid leukemia cells.
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Author keywords
[No Author keywords available]
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Indexed keywords
BUTYRIC ACID DERIVATIVE;
CD11 ANTIGEN;
CD18 ANTIGEN;
ENZYME INHIBITOR;
HISTONE DEACETYLASE;
I KAPPA B;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
NF KAPPAB INHIBITOR ALPHA;
NF-KAPPAB INHIBITOR ALPHA;
P21 ACTIVATED KINASE;
PROTEIN SERINE THREONINE KINASE;
TUMOR NECROSIS FACTOR ALPHA;
APOPTOSIS;
ARTICLE;
CELL STRAIN U937;
DRUG EFFECT;
GENE EXPRESSION REGULATION;
HUMAN;
METABOLISM;
PHYSIOLOGY;
PROMOTER REGION;
PROTEIN PROCESSING;
SIGNAL TRANSDUCTION;
ANTIGENS, CD11;
ANTIGENS, CD18;
APOPTOSIS;
BUTYRATES;
ENZYME INHIBITORS;
GENE EXPRESSION REGULATION;
HISTONE DEACETYLASES;
HUMANS;
I-KAPPA B PROTEINS;
NF-KAPPA B;
PROMOTER REGIONS (GENETICS);
PROTEIN PROCESSING, POST-TRANSLATIONAL;
PROTEIN-SERINE-THREONINE KINASES;
SIGNAL TRANSDUCTION;
TUMOR NECROSIS FACTOR-ALPHA;
U937 CELLS;
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EID: 1642576624
PISSN: 15384101
EISSN: None
Source Type: Journal
DOI: 10.4161/cc.2.5.465 Document Type: Article |
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Times cited : (31)
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References (0)
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