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Volumn 20, Issue 2, 2003, Pages 123-129
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Interaction between the innate and adaptive immune systems is required to survive sepsis and control inflammation after injury.
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Author keywords
[No Author keywords available]
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Indexed keywords
CYTOKINE;
HOMEODOMAIN PROTEIN;
INTERLEUKIN 6;
LIPOPOLYSACCHARIDE;
RAG 1 PROTEIN;
RAG-1 PROTEIN;
TUMOR NECROSIS FACTOR ALPHA;
ANIMAL;
ARTICLE;
B LYMPHOCYTE;
BIOSYNTHESIS;
BURN;
C57BL MOUSE;
CELL SEPARATION;
CYTOLOGY;
FLOW CYTOMETRY;
GENETICS;
IMMUNOLOGY;
INFLAMMATION;
MACROPHAGE;
MALE;
METABOLISM;
MOUSE;
PATHOLOGY;
SEPSIS;
SPLEEN;
T LYMPHOCYTE;
TIME;
TRANSGENIC MOUSE;
ANIMALS;
B-LYMPHOCYTES;
BURNS;
CELL SEPARATION;
CYTOKINES;
FLOW CYTOMETRY;
HOMEODOMAIN PROTEINS;
INFLAMMATION;
INTERLEUKIN-6;
LIPOPOLYSACCHARIDES;
MACROPHAGES;
MALE;
MICE;
MICE, INBRED C57BL;
MICE, TRANSGENIC;
SEPSIS;
SPLEEN;
T-LYMPHOCYTES;
TIME FACTORS;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 1542308211
PISSN: 10732322
EISSN: None
Source Type: Journal
DOI: 10.1097/01.shk.0000079426.52617.00 Document Type: Article |
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Times cited : (87)
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References (0)
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