Intact nitric oxide production is obligatory for the sustained flow response during hypercapnic acidosis in guinea pig heart

Cardiovascular Research

Volumn 66, Issue 1, 2005, Pages 55-63

  Heintz, Anke ^a   Koch, Thea ^a   Deussen, Andreas ^b  

^a UNIVERSITY HOSPITAL CARL GUSTAV CARUS   (Germany)

^b DRESDEN UNIVERSITY OF TECHNOLOGY   (Germany)

Author keywords

Acidosis; Coronary circulation; Hypercapnia; K+ATP channel; Nitric oxide; Vasoconstriction dilation

Indexed keywords

CYCLIC GMP; GLIBENCLAMIDE; N(G) METHYLARGININE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; POTASSIUM CHANNEL BLOCKING AGENT;

ANIMAL TISSUE; ARTERIAL CARBON DIOXIDE TENSION; ARTICLE; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; DRUG EFFECT; FEMALE; HEART PERFUSION; HYPERCAPNIA; HYPEREMIA; MALE; NONHUMAN; PERFUSION PRESSURE; PRIORITY JOURNAL; RESPIRATORY ACIDOSIS;

ACIDOSIS; ANIMALS; CORONARY CIRCULATION; ENDOTHELIUM, VASCULAR; FEMALE; GLYBURIDE; GUINEA PIGS; HYPERCAPNIA; MALE; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; OMEGA-N-METHYLARGININE; OXYGEN CONSUMPTION; PERFUSION; POTASSIUM CHANNEL BLOCKERS; POTASSIUM CHANNELS; TIME FACTORS; VASODILATION;

EID: 14844354513     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.cardiores.2004.12.001     Document Type: Article

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