Vitamin D Enhances Caspase-Dependent and Independent TNF-Induced Breast Cancer Cell Death: The Role of Reactive Oxygen Species

Annals of the New York Academy of Sciences

Volumn 1010, Issue , 2003, Pages 437-440

  Weitsman, G E ^a   Ravid, A ^a   Liberman, U A ^a   Koren, R ^a  

^a TEL AVIV UNIVERSITY   (Israel)

Author keywords

Calcitriol; Mitochondria; Reactive oxygen species; TNF

Indexed keywords

ACETYLCYSTEINE; ANTIOXIDANT; ASCORBIC ACID; CALCITRIOL; CASPASE INHIBITOR; CYTOKINE; FLUORESCENT DYE; GLUTATHIONE; REACTIVE OXYGEN METABOLITE; THIOCTIC ACID; TUMOR NECROSIS FACTOR ALPHA;

BREAST CANCER; CANCER CELL; CELL DEATH; CONFERENCE PAPER; CONTROLLED STUDY; DRUG CYTOTOXICITY; ENZYME ACTIVATION; ENZYME ACTIVITY; ENZYME INDUCTION; HORMONE ACTION; HUMAN; HUMAN CELL; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; PROTEIN INTERACTION;

MINK CELL FOCUS-FORMING VIRUS;

EID: 1342347862     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1299.079     Document Type: Conference Paper

References (5)

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4. LEIST M. & M. JAATTELA. 2001. Four deaths and a funeral: from caspases to alternative mechanisms. Nat. Rev. Mol. Cell. Biol. 2: 589-598
5. FLEURY C., B. MIGNOTTE & J.L. VAYSSIERE. 2002. Mitochondrial reactive oxygen species in cell death signaling. Biochimie 84: 131-141.