Oxidative stress-induced apoptosis is associated with alterations in mitochondrial caspase activity and Bcl-2-dependent alterations in mitochondrial pH (pHm)

Brain Research Bulletin

Volumn 62, Issue 6, 2004, Pages 497-504

  Takahashi, Akiyuki ^a,b   Masuda, Atsushi ^a   Sun, Mao ^a,c   Centonze, Victoria E ^a   Herman, Brian ^a  

^a Mays Cancer Center at UT Health San Antonio   (United States)

^b KYOTO PREFECTURAL UNIVERSITY   (Japan)

^c TEXAS TECH UNIVERSITY HEALTH SCIENCES CENTER SCHOOL OF MEDICINE   (United States)

Author keywords

Mitochondrial; Opthamological diseases; pH

Indexed keywords

ADENOSINE TRIPHOSPHATE; CASPASE 2; CASPASE 9; CYTOCHROME C; OXIDOREDUCTASE; PROTEIN BCL 2; PROTEIN DERIVATIVE; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;

ACIDIFICATION; ACIDITY; ALKALINITY; APOPTOSIS; CONFERENCE PAPER; ENZYME ACTIVATION; ENZYME ACTIVITY; HUMAN; IMMUNOPRECIPITATION; MITOCHONDRIAL RESPIRATION; MOLECULAR MECHANICS; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN LOCALIZATION; PROTEIN PROTEIN INTERACTION; PROTEIN SECRETION; PROTEIN SYNTHESIS; REGULATORY MECHANISM; TISSUE INJURY;

EID: 1342333859     PISSN: 03619230     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.brainresbull.2003.07.009     Document Type: Conference Paper

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