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Volumn 18, Issue 1, 2004, Pages 164-166
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Glucocorticoid receptor deficiency increases vulnerability of the nigrostriatal dopaminergic system: critical role of glial nitric oxide.
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Author keywords
[No Author keywords available]
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Indexed keywords
CORTICOSTERONE;
DOPAMINE;
DRUG DERIVATIVE;
ENZYME INHIBITOR;
GLUCOCORTICOID RECEPTOR;
INDUCIBLE NITRIC OXIDE SYNTHASE;
LYSINE;
N(6) (1 IMINOETHYL)LYSINE;
N(6)-(1-IMINOETHYL)LYSINE;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
NOS2 PROTEIN, MOUSE;
TYROSINE 3 MONOOXYGENASE;
ANIMAL;
ARTICLE;
DRUG ANTAGONISM;
DRUG EFFECT;
ENZYMOLOGY;
GENETICS;
GLIA;
INTOXICATION;
MACROPHAGE;
METABOLISM;
MOUSE;
NEOSTRIATUM;
NERVE CELL;
OXIDATIVE STRESS;
PATHOLOGY;
PHYSIOLOGY;
SUBSTANTIA NIGRA;
TRANSGENIC MOUSE;
ANIMALS;
CORTICOSTERONE;
DOPAMINE;
ENZYME INHIBITORS;
LYSINE;
MACROPHAGES;
MICE;
MICE, TRANSGENIC;
MPTP POISONING;
NEOSTRIATUM;
NEUROGLIA;
NEURONS;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
NITRIC OXIDE SYNTHASE TYPE II;
OXIDATIVE STRESS;
RECEPTORS, GLUCOCORTICOID;
SUBSTANTIA NIGRA;
TYROSINE 3-MONOOXYGENASE;
MLCS;
MLOWN;
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EID: 1342332143
PISSN: None
EISSN: 15306860
Source Type: Journal
DOI: 10.1096/fj.03-0501fje Document Type: Article |
Times cited : (65)
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References (0)
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