Proteolytic Activation of Proapoptotic Kinase PKCδ Is Regulated by Overexpression of Bcl-2: Implications for Oxidative Stress and Environmental Factors in Parkinson's Disease

Annals of the New York Academy of Sciences

Volumn 1010, Issue , 2003, Pages 683-686

  Kanthasamy, A G ^a   Kitazawa, M ^a   Kaul, S ^a   Yang, Y ^a   Lahiri, D K ^b   Anantharam, V ^a   Kanthasamy, A ^a  

^a IOWA STATE UNIVERSITY   (United States)

^b INDIANA UNIVERSITY   (United States)

Author keywords

Apoptosis; Dieldrin; Dopaminergic degeneration; Environmental factors; Oxidative stress; Pesticide; Protein kinase C

Indexed keywords

CASPASE 3; DIELDRIN; DNA FRAGMENT; MITOCHONDRIAL PROTEIN; PROTEIN BCL 2; PROTEIN KINASE C DELTA;

APOPTOSIS; CONFERENCE PAPER; DOPAMINERGIC NERVE CELL; DOSE RESPONSE; ENZYME ACTIVATION; ENZYME REGULATION; GENE OVEREXPRESSION; HUMAN; MITOCHONDRION; NERVE DEGENERATION; OXIDATION REDUCTION REACTION; PROTEIN DEGRADATION; PROTEIN EXPRESSION; PROTEIN FUNCTION;

EID: 1342305460     PISSN: 00778923     EISSN: None     Source Type: Book Series    
DOI: 10.1196/annals.1299.125     Document Type: Conference Paper

References (5)

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4. ANANTHARAM, V., M. KITAZAWA, J. WAGNER, et al. 2002. Caspase-3-dependent proteolytic cleavage of protein kinase Cdelta is essential for oxidative stress-mediated dopaminergic cell death after exposure to methylcyclopentadienyl manganese tricarbonyl. J. Neurosci. 22: 1738-1751.
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