Melatonin-induced neuroprotection after closed head injury is associated with increased brain antioxidants and attenuated late-phase activation of NF-kappaB and AP-1.

The FASEB journal : official publication of the Federation of American Societies for Experimental Biology

Volumn 18, Issue 1, 2004, Pages 149-151

  Beni, Sara M ^a   Kohen, Ron ^a   Reiter, Russel J ^a   Tan, Dun Xian ^a   Shohami, Esther ^a  

^a HEBREW UNIVERSITY OF JERUSALEM   (Israel)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; MELATONIN; NEUROPROTECTIVE AGENT; TRANSCRIPTION FACTOR AP 1;

ANIMAL; ARTICLE; BRAIN; DRUG EFFECT; HEAD INJURY; KINETICS; METABOLISM; MOUSE; OXIDATION REDUCTION REACTION;

ANIMALS; ANTIOXIDANTS; BRAIN; HEAD INJURIES, CLOSED; KINETICS; MELATONIN; MICE; NEUROPROTECTIVE AGENTS; NF-KAPPA B; OXIDATION-REDUCTION; TRANSCRIPTION FACTOR AP-1;

MLCS; MLOWN;

EID: 1342268351     PISSN: None     EISSN: 15306860     Source Type: Journal    
DOI: 10.1096/fj.03-0323fje     Document Type: Article

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