Bimodal temporal distribution of herpetic reactivation [3]

Mayo Clinic Proceedings

Volumn 80, Issue 2, 2005, Pages 287-288

  Burkhart, Craig N ^a   Burkhart, Craig G ^b  

^a UNIVERSITY OF NORTH CAROLINA   (United States)

^b MEDICAL COLLEGE OF OHIO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ACICLOVIR; ANTIVIRUS AGENT; VALACICLOVIR;

EPIDERMIS CELL; HERPES LABIALIS; HERPES SIMPLEX VIRUS; HUMAN; LETTER; NONHUMAN; PATHOGENESIS; PERIPHERAL NERVE; PROPHYLAXIS; RECURRENT DISEASE; SENSORY NERVE; SPINAL GANGLION; ULTRAVIOLET RADIATION; VIRUS GENOME; VIRUS REACTIVATION; VIRUS REPLICATION;

EID: 13244271277     PISSN: 00256196     EISSN: None     Source Type: Journal    
DOI: 10.4065/80.2.287-b     Document Type: Letter

References (6)

1. Spruance SL, Freeman DJ, Stewart JC, et al. The natural history of ultraviolet radiation-induced herpes simplex labialis and response to therapy with peroral and topical formulations of acyclovir. J Infect Dis. 1991;163:728-734.
2. Spruance SL, Kriesel JD, Evans TG, McKeough MB. Susceptibility to herpes labialis following multiple experimental exposures to ultraviolet radiation. Antiviral Res. 1995;28:57-67.
3. Maratou E, Theophilidis G, Arsenakis M. Axonal transport of herpes simplex virus-1 in an in vitro model based on the isolated sciatic nerve of the frog Rana ridibunda. J Neurosci Methods. 1998;79:75-78.
4. Miura S, Smith CC, Burnett JW, Aurelian L. Detection of viral DNA within skin of healed recurrent herpes simplex infection and erythema multiforme lesions. J Invest Dermatol. 1992;98:68-72.
5. Mertz GJ, Jones CC, Mills J, et al. Long-term acyclovir suppression of frequently recurring genital herpes simplex virus infection: a multicenter double-blind trial. JAMA. 1988;260:201-206.
6. Corey L. Suppressive therapy: reduction of risk of transmission of virus to partner. In: Program and Abstracts of the 42nd Interscience Conference on Antimi-crobial Agents and Chemotherapy; San Diego, Calif; September 27-30, 2002. Abstract 39.