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Volumn 36, Issue 1, 2005, Pages 39-42

Effect of tyrosine kinase inhibitors on airway remodeling in bronchial asthma

Author keywords

Airway remodeling; Epidermal growth factor receptor (EGFR); Tyrosine kinase inhibitor

Indexed keywords

4 (3 CHLOROANILINO) 6,7 DIMETHOXYQUINAZOLINE; COLLAGEN TYPE 1; COLLAGEN TYPE 3; EPIDERMAL GROWTH FACTOR RECEPTOR; GENISTEIN; JIN ZHUAN TING; PROTEIN TYROSINE KINASE INHIBITOR; UNCLASSIFIED DRUG;

EID: 12744273675     PISSN: 1672173X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (3)

References (9)
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  • 2
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    • Bronchial epithelium as a key regulator of airway allergen sensitization and remodeling in asthma
    • Holgate ST, Lackie P, Wilson S, et al. Bronchial epithelium as a key regulator of airway allergen sensitization and remodeling in asthma. Am J Respir Crit Care Med, 2000;162(3 Pt 2):s113.
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  • 5
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    • Asthma: A disease remodeling the airways
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  • 7
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    • 4-Hydroxy-2-nonenal enhances fibronectin production by IMP-90 human lung fibroblasts partly via activation of epidermal growth factor receptor-linked extracellular signal-regulated kinase p44/42 pathway
    • Tsukagoshi H, Kawata T, Shimizu Y, et al. 4-Hydroxy-2-nonenal enhances fibronectin production by IMP-90 human lung fibroblasts partly via activation of epidermal growth factor receptor-linked extracellular signal-regulated kinase p44/42 pathway. Toxicol Appl Pharmacol, 2002;184(3):127.
    • (2002) Toxicol Appl Pharmacol , vol.184 , Issue.3 , pp. 127
    • Tsukagoshi, H.1    Kawata, T.2    Shimizu, Y.3
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.