IL-6-deficient mice are susceptible to ethanol-induced hepatic steatosis: IL-6 protects against ethanol-induced oxidative stress and mitochondrial permeability transition in the liver.

Cellular & molecular immunology

Volumn 1, Issue 3, 2004, Pages 205-211

  El Assal, Osama ^a   Hong, Feng ^a   Kim, Won Ho ^a   Radaeva, Svetlana ^a   Gao, Bin ^a  

^a NATIONAL INSTITUTE ON ALCOHOL ABUSE AND ALCOHOLISM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALCOHOL; INTERLEUKIN 6; SOLVENT;

ALCOHOL LIVER DISEASE; ANIMAL; ANIMAL HEPATITIS; ARTICLE; CHEMICALLY INDUCED DISORDER; DRUG EFFECT; GENETICS; HUMAN; LIVER; LIVER CELL; LIVER MITOCHONDRION; METABOLISM; MOUSE; MOUSE MUTANT; OXIDATIVE STRESS; PATHOLOGY; RAT;

ANIMALS; ETHANOL; HEPATITIS, ALCOHOLIC; HEPATITIS, ANIMAL; HEPATOCYTES; HUMANS; INTERLEUKIN-6; LIVER; MICE; MICE, KNOCKOUT; MITOCHONDRIA, LIVER; OXIDATIVE STRESS; RATS; SOLVENTS;

EID: 12744259191     PISSN: 16727681     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (0)