Intracellular acidosis-activated p38 MAPK signaling and its essential role in cardiomyocyte hypoxic injury

FASEB Journal

Volumn 19, Issue 1, 2005, Pages 109-111

  Zheng, Ming ^a,b   Reynolds, Cherilynn ^c   Jo, Su Hyun ^c,e   Wersto, Robert ^d   Han, Qide ^a   Xiao, Rui Ping ^a,b,d  

^a Cardiology Division   (United States)

^b PEKING UNIVERSITY   (China)

^c Laboratory of Cardiovascular Science   (United States)

^d NATIONAL INSTITUTE ON AGING   (United States)

^e Jeju National University School of Medicine   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE KINASE 3; MITOGEN ACTIVATED PROTEIN KINASE P38;

ACIDOSIS; ANIMAL CELL; ANIMAL MODEL; ARTICLE; CALCIUM CELL LEVEL; CALCIUM SIGNALING; CELL DEATH; CELL PROTECTION; ENZYME ACTIVATION; ENZYME INHIBITION; HEART MUSCLE INJURY; HYPOXIA; NONHUMAN; PH; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; RAT; SIGNAL TRANSDUCTION; WESTERN BLOTTING;

ACIDOSIS; ANIMALS; ANOXIA; CALCIUM SIGNALING; ENZYME ACTIVATION; HEART VENTRICLES; HYDROGEN-ION CONCENTRATION; MAP KINASE KINASE 3; MAP KINASE KINASE 6; MYOCYTES, CARDIAC; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; RATS; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 11244305854     PISSN: 08926638     EISSN: None     Source Type: Journal    
DOI: 10.1096/fj.04-2607fje     Document Type: Article

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