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Volumn 18, Issue 15, 2004, Pages 1906-1908
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Cyclic exposure to hypoxia and reoxygenation selects for tumor cells with defects in mitochondrial apoptotic pathways
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Author keywords
[No Author keywords available]
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Indexed keywords
CASPASE;
ETOPOSIDE;
HYPOXIA INDUCIBLE FACTOR 1ALPHA;
PROTEIN BAX;
PROTEIN P21;
PROTEIN P53;
REGULATOR PROTEIN;
TUMOR NECROSIS FACTOR RELATED APOPTOSIS INDUCING LIGAND;
APOPTOSIS;
ARTICLE;
CONTROLLED STUDY;
ENZYME ACTIVATION;
HUMAN;
HUMAN CELL;
HYPOXIA;
IONIZING RADIATION;
MITOCHONDRION;
PRIORITY JOURNAL;
PROTEIN CONFORMATION;
PROTEIN EXPRESSION;
PROTEIN FUNCTION;
RADIATION INJURY;
REOXYGENATION;
SIGNAL TRANSDUCTION;
TUMOR CELL;
APOPTOSIS;
APOPTOSIS REGULATORY PROTEINS;
BCL-2-ASSOCIATED X PROTEIN;
CELL CYCLE PROTEINS;
CELL HYPOXIA;
CELL LINE, TUMOR;
CELL RESPIRATION;
CYCLIN-DEPENDENT KINASE INHIBITOR P21;
ETOPOSIDE;
HUMANS;
HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT;
MEMBRANE GLYCOPROTEINS;
MITOCHONDRIA;
NEOPLASMS;
PERIODICITY;
PROTO-ONCOGENE PROTEINS C-BCL-2;
RADIATION, IONIZING;
SIGNAL TRANSDUCTION;
TNF-RELATED APOPTOSIS-INDUCING LIGAND;
TRANSCRIPTION FACTORS;
TUMOR NECROSIS FACTOR-ALPHA;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 10044277941
PISSN: 08926638
EISSN: None
Source Type: Journal
DOI: 10.1096/fj.04-1918fje Document Type: Article |
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Times cited : (58)
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References (0)
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