Antiproliferative signalling by 1,25(OH)2D3 in prostate and breast cancer is suppressed by a mechanism involving histone deacetylation.

Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer

Volumn 164, Issue , 2003, Pages 83-98

  Banwell, Claire M ^a   Singh, Rena ^a   Stewart, Paul M ^a   Uskokovic, Milan R ^a   Campbell, Moray J ^a  

^a UNIVERSITY DEPARTMENT OF MEDICINE   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

19 NOR HEXAFLUORIDE VITAMIN D3; 19-NOR-HEXAFLUORIDE VITAMIN D3; ANDROGEN; ANTINEOPLASTIC AGENT; COLECALCIFEROL; DIHYDROXY VITAMIN D3; DIHYDROXY-VITAMIN D3; DNA BINDING PROTEIN; DRUG DERIVATIVE; ENZYME INHIBITOR; HISTONE DEACETYLASE; HYDROXAMIC ACID; LIGAND; NUCLEAR RECEPTOR CO REPRESSOR 2; NUCLEAR RECEPTOR CO-REPRESSOR 2; REPRESSOR PROTEIN; TRICHOSTATIN A; VITAMIN D;

ARTICLE; BREAST TUMOR; CELL CULTURE; CELL DIVISION; DRUG ANTAGONISM; DRUG EFFECT; DRUG POTENTIATION; HUMAN; MALE; METABOLISM; NEOPLASM; PATHOLOGY; PHYSIOLOGY; PROSTATE TUMOR;

ANDROGENS; ANTINEOPLASTIC AGENTS; BREAST NEOPLASMS; CELL DIVISION; CHOLECALCIFEROL; DNA-BINDING PROTEINS; DRUG SYNERGISM; ENZYME INHIBITORS; HISTONE DEACETYLASES; HUMANS; HYDROXAMIC ACIDS; LIGANDS; MALE; NEOPLASMS, HORMONE-DEPENDENT; PROSTATIC NEOPLASMS; REPRESSOR PROTEINS; TUMOR CELLS, CULTURED; VITAMIN D;

EID: 0642336667     PISSN: 00800015     EISSN: None     Source Type: Journal    
DOI: 10.1007/978-3-642-55580-0_5     Document Type: Article

References (0)