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Volumn 17, Issue 15, 2003, Pages 2266-2268
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Complement C3a receptors in the pituitary gland: A novel pathway by which an innate immune molecule releases hormones involved in the control of inflammation
b
UWCM
(United Kingdom)
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Author keywords
[No Author keywords available]
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Indexed keywords
COMPLEMENT COMPONENT C3 RECEPTOR;
COMPLEMENT COMPONENT C3A;
COMPLEMENT COMPONENT C3A RECEPTOR;
G PROTEIN COUPLED RECEPTOR;
UNCLASSIFIED DRUG;
COMPLEMENT C3A RECEPTOR;
COMPLEMENT C3A, DES ARG (77);
COMPLEMENT C3A, DES-ARG-(77);
COMPLEMENT RECEPTOR;
HYPOPHYSIS HORMONE;
MEMBRANE PROTEIN;
PERTUSSIS TOXIN;
PLASMA PROTEIN;
ANTIINFLAMMATORY ACTIVITY;
ARTICLE;
CENTRAL NERVOUS SYSTEM;
COMPLEMENT ACTIVATION;
CONTROLLED STUDY;
CYTOKINE RELEASE;
HORMONE RELEASE;
HUMAN;
HUMAN CELL;
HYPOPHYSIS FUNCTION;
HYPOTHALAMUS HYPOPHYSIS ADRENAL SYSTEM;
IMMUNE RESPONSE;
IMMUNE SYSTEM;
INFLAMMATION;
PRIORITY JOURNAL;
PROTEIN ANALYSIS;
RECEPTOR AFFINITY;
RECEPTOR UPREGULATION;
ANIMAL;
BIOLOGICAL MODEL;
CELL CULTURE;
DRUG EFFECT;
ENDOCRINE SYSTEM;
HYPOPHYSIS;
IMMUNOLOGY;
INNATE IMMUNITY;
METABOLISM;
RAT;
SECRETION;
SIGNAL TRANSDUCTION;
ANIMALS;
BLOOD PROTEINS;
CELLS, CULTURED;
COMPLEMENT C3A;
ENDOCRINE SYSTEM;
IMMUNITY, NATURAL;
INFLAMMATION;
MEMBRANE PROTEINS;
MODELS, IMMUNOLOGICAL;
PERTUSSIS TOXIN;
PITUITARY GLAND;
PITUITARY HORMONES;
RATS;
RECEPTORS, COMPLEMENT;
SIGNAL TRANSDUCTION;
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EID: 0642333822
PISSN: 08926638
EISSN: None
Source Type: Journal
DOI: 10.1096/fj.02-1103fje Document Type: Article |
Times cited : (58)
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References (0)
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