The cellular immunodeficiency associated with post-traumatic stress disorder may be the result of sympathetic overactivity and be correctable by beta-2-blockers

Neuroendocrinology Letters

Volumn 24, Issue 6, 2003, Pages 468-

  Namazi, M R ^a,b  

^a SHIRAZ UNIVERSITY OF MEDICAL SCIENCES   (Iran)

^b NONE   (Iran)

Author keywords

[No Author keywords available]

Indexed keywords

BETA 2 ADRENERGIC RECEPTOR BLOCKING AGENT; NORADRENALIN;

ANTIGEN EXPRESSION; AROUSAL; CATECHOLAMINE RELEASE; CELLULAR IMMUNODEFICIENCY; CYTOKINE RELEASE; DRUG MECHANISM; HUMAN; LETTER; NORADRENALIN URINE LEVEL; POSTTRAUMATIC STRESS DISORDER; SYMPATHETIC TONE; T LYMPHOCYTE SUBPOPULATION; TH2 CELL;

EID: 0347692877     PISSN: 0172780X     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Letter

References (5)

1. Editorial. Stress and the skin. Arch Dermatol 2001; 137:78-82
2. Wong CM. Post-traumatic stress disorder: Advances in psychoneuroimmunology. Psychiatr Clin N Am 2002; 25: 369-83.
3. Kawamura N, Kim Y, Asukai N. Suppression of cellular immunity in men with a past history of post-traumatic stress disorder. Am J Psychiatry 2001; 158:484-6.
4. Kaplan HL, Sadock BJ. Synopsis of Psychiatry. 6th edn. Baltimore: Williams and Wilkins. 1991. pp. 409-12.
5. Oberbeck R, Van Griensven M, Nickel E, Tschernign T, Wittwer T, Tape HC. Influence of beta-adrenergic antagonists on hemorrhage-induced cellular immune suppression. Shock 2002; 18(4): 331-5.