Shock-induced arrhythmogenesis is enhanced by 2,3-butanedione monoxime compared with cytochalasin D

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 286, Issue 1 55-1, 2004, Pages

  Cheng, Yuanna ^a   Li, Li ^b   Nikolski, Vladimir ^b   Wallick, Don W ^a   Efimov, Igor R ^a  

^a LERNER RESEARCH INSTITUTE   (United States)

^b Case Western Reserve University   (United States)

Author keywords

Action potential duration restitution; Cardiac vulnerability; Excitation contraction uncouplers; Optical mapping

Indexed keywords

CYTOCHALASIN D; DIACETYL OXIME;

ACTION POTENTIAL; ANIMAL TISSUE; ARRHYTHMOGENESIS; ARTICLE; CLINICAL PROTOCOL; CONTROLLED STUDY; ELECTRIC ACTIVITY; ELECTRIC SHOCK; EPICARDIUM MAPPING; EXCITATION CONTRACTION COUPLING; HEART; HEART ARRHYTHMIA; HEART REPOLARIZATION; HEART VENTRICLE TACHYCARDIA; ISOLATED HEART; MONOMORPHIC VENTRICULAR TACHYCARDIA; NONHUMAN; OPTICAL MAPPING; POLYMORPHIC VENTRICULAR TACHYCARDIA; PRIORITY JOURNAL; RABBIT;

EID: 0346998126     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00092.2003     Document Type: Article

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