Increased amphetamine-induced locomotion during inactivation of the basolateral amygdala

Behavioural Brain Research

Volumn 149, Issue 1, 2004, Pages 33-39

  Woods, Vanessa Erin ^a   Ettenberg, Aaron ^a  

^a UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Dopamine; Lidocaine; Locomotion; Nucleus accumbens; Rat; Reversible lesions

Indexed keywords

AMPHETAMINE; LIDOCAINE;

AMYGDALOID NUCLEUS; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN INJURY; CELL FUNCTION; CONTROLLED STUDY; DOPAMINERGIC NERVE CELL; DRUG EFFECT; DRUG MECHANISM; HYPERACTIVITY; LIMBIC CORTEX; LOCOMOTION; MALE; NERVE ENDING; NONHUMAN; NUCLEUS ACCUMBENS; PRIORITY JOURNAL; RAT; REGULATORY MECHANISM; RELIABILITY;

EID: 0346671127     PISSN: 01664328     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0166-4328(03)00212-2     Document Type: Article

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