The Aγ-195 (C→G) mutation in hereditary persistence of fetal hemoglobin is not associated with activation of a reporter gene in vitro

Brazilian Journal of Medical and Biological Research

Volumn 34, Issue 4, 2001, Pages 489-492

  Schreiber, R ^a   Goncalves M S ^a   Junqueira, M L ^b   Saad, S T O ^a   Krieger, J E ^b   Costa, F F ^a  

^a UNIVERSITY OF CAMPINAS   (Brazil)

^b UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Fetal hemoglobin; Hereditary persistence of fetal hemoglobin; HPFH; Transient expression

Indexed keywords

BETA GALACTOSIDASE; GLOBIN; HEMOGLOBIN F; LUCIFERASE; PRIMER DNA;

ADULT; ARTICLE; GENE EXPRESSION; GENETIC TRANSFECTION; GENETICS; HEMOGLOBINOPATHY; HUMAN; METABOLISM; MUTATION; POINT MUTATION; POLYMERASE CHAIN REACTION; REPORTER GENE;

ADULT; BETA-GALACTOSIDASE; DNA PRIMERS; FETAL HEMOGLOBIN; GENE EXPRESSION; GENES, REPORTER; GLOBINS; HEMOGLOBINOPATHIES; HUMANS; LUCIFERASES; MUTATION; POINT MUTATION; POLYMERASE CHAIN REACTION; TRANSFECTION;

EID: 0346511440     PISSN: 0100879X     EISSN: 1414431X     Source Type: Journal    
DOI: 10.1590/S0100-879X2001000400008     Document Type: Article

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