Activation of Ca2+-dependent K+ channels contributes to rhythmic firing of action potentials in mouse pancreatic β cells

Journal of General Physiology

Volumn 114, Issue 6, 1999, Pages 759-769

  Göpel, Sven O ^a   Kanno, Takahiro ^a   Barg, Sebastian ^a   Eliasson, Lena ^a   Galvanovskis, Juris ^a   Renström, Erik ^a   Rorsman, Patrik ^a  

^a LUND UNIVERSITY   (Sweden)

Author keywords

Ca2+; Ca2+ activated K+ channel; Insulin; Membrane potential; Pancreas

Indexed keywords

APAMIN; CALCIUM; GLUCOSE; INSULIN; POTASSIUM; POTASSIUM CHANNEL;

ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; CELL ACTIVATION; CELL MEMBRANE DEPOLARIZATION; ELECTRIC CURRENT; MEMBRANE POTENTIAL; MOUSE; NONHUMAN; PANCREAS ISLET BETA CELL; POTASSIUM CONDUCTANCE;

ACTION POTENTIALS; ANIMALS; ATP-BINDING CASSETTE TRANSPORTERS; ELECTROPHYSIOLOGY; HYPOGLYCEMIC AGENTS; ISLETS OF LANGERHANS; LARGE-CONDUCTANCE CALCIUM-ACTIVATED POTASSIUM CHANNELS; MEMBRANE POTENTIALS; MICE; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNEL BLOCKERS; POTASSIUM CHANNELS; POTASSIUM CHANNELS, CALCIUM-ACTIVATED; POTASSIUM CHANNELS, INWARDLY RECTIFYING; TOLBUTAMIDE;

EID: 0344339740     PISSN: 00221295     EISSN: None     Source Type: Journal    
DOI: 10.1085/jgp.114.6.759     Document Type: Article

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