The c-Myc oncoprotein interacts with Bcr

Current Biology

Volumn 13, Issue 5, 2003, Pages 437-441

  Mahon, Gwendolyn M ^a   Wang, Yan ^b   Korus, Malgorzata ^a   Kostenko, Elena ^a   Cheng, Li ^a   Sun, Tong ^b   Arlinghaus, Ralph B ^b   Whitehead, Ian P ^a  

^a RUTGERS NEW JERSEY MEDICAL SCHOOL   (United States)

^b UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BCR PROTEIN, HUMAN; BREAKPOINT CLUSTER REGION PROTEIN; MYC PROTEIN; ONCOPROTEIN; PROTEIN TYROSINE KINASE;

ANIMAL; ARTICLE; HUMAN; METABOLISM; YEAST;

ANIMALS; HUMANS; ONCOGENE PROTEINS; PROTEIN-TYROSINE KINASES; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCR; PROTO-ONCOGENE PROTEINS C-MYC; YEASTS;

ANIMALIA; MAMMALIA;

EID: 0242501528     PISSN: 09609822     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0960-9822(03)00090-3     Document Type: Article

References (12)

1. Solomon, D.L., Amati, B., and Land, H. (1993). Distinct DNA binding preferences for the c-Myc/Max and Max/Max dimers. Nucleic Acids Res. 21, 5372-5376.
2. Prendergast, G.C., and Ziff, E.B. (1991). Methylation-sensitive sequence-specific DNA binding by the c-Myc basic region. Science 251, 186-189.
3. Blackwell, T.K., and Weintraub, H. (1990). Differences and similarities in DNA-binding preferences of MyoD and E2A protein complexes revealed by binding site selection. Science 250, 1104-1110.
4. Claassen, G.F., and Hann, S.R. (1999). Myc-mediated transformation: the repression connection. Oncogene 18, 2925-2933.
5. Bouchard, C., Thieke, K., Maier, A., Saffrich, R., Hanley-Hyde, J., Ansorge, W., Reed, S., Sicinski, P., Bartek, J., and Eilers, M. (1999). Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27. EMBO J. 18, 5321-5333.
6. Land, H., Parada, L.F., and Weinberg, R.A. (1983). Tumorigenic conversion of primary embryo fibroblasts requires at least two cooperating oncogenes. Nature 304, 596-602.
7. Ruley, H. E. (1990). Transforming collaborations between ras and nuclear oncogenes. Cancer Cells 2, 258-268.
8. Grandori, C., Cowley, S.M., James, L.P., and Eisenman, R.N. (2000). The Myc/Max/Mad network and the transcriptional control of cell behavior. Annu. Rev. Cell Dev. Biol. 16, 653-699.
9. Lin, F., Monaco, G., Sun, T., Liu, J., Lin, H., Stephens, C., Belmont, J., and Arlinghaus, R.B. (2001). BCR gene expression blocks Bcr-Abl induced pathogenicity in a mouse model. Oncogene 20, 1873-1881.
10. Nesbit, C.E., Tersak, J.M., and Prochownik, E.V. (1999). MYC oncogenes and human neoplastic disease. Oncogene 18, 3004-3016.
11. Sawyers, C.L. (1993). The role of myc in transformation by BCR-ABL. Leuk. Lymphoma 11, 45-46.
12. Sawyers, C.L., Callahan, W., and Witte O.N. (1992). Dominant negative MYC blocks transformation by ABL oncogenes. Cell 70, 901-910.