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Volumn 100, Issue 22, 2003, Pages 12935-12940
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The development of diabetes in E2f1/E2f2 mutant mice reveals important roles for bone marrow-derived cells in preventing islet cell loss
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Author keywords
[No Author keywords available]
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Indexed keywords
RECOMBINANT HUMAN INSULIN;
TRANSCRIPTION FACTOR E2F;
TRANSCRIPTION FACTOR E2F1;
TRANSCRIPTION FACTOR E2F2;
UNCLASSIFIED DRUG;
AGE;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
BONE MARROW CELL;
BONE MARROW TRANSPLANTATION;
CELL CYCLE;
CELL FUNCTION;
CELL LOSS;
CONTROLLED STUDY;
EXOCRINE CELL;
FEMALE;
HEMATOPOIETIC SYSTEM;
HUMAN;
INSULIN DEPENDENT DIABETES MELLITUS;
MALE;
MOUSE;
MUTANT;
NONHUMAN;
PANCREAS;
PANCREAS DISEASE;
PANCREAS ISLET BETA CELL;
PANCREAS ISLET CELL;
PANCREATIC DEGENERATION;
PENETRANCE;
PRIORITY JOURNAL;
REGULATORY MECHANISM;
STRESS;
WILD TYPE;
ANIMALS;
BLOOD GLUCOSE;
BONE MARROW CELLS;
BONE MARROW TRANSPLANTATION;
CELL CYCLE PROTEINS;
DIABETES MELLITUS;
DNA-BINDING PROTEINS;
E2F TRANSCRIPTION FACTORS;
E2F1 TRANSCRIPTION FACTOR;
FEMALE;
INSULIN;
ISLETS OF LANGERHANS;
MALE;
MICE;
MICE, KNOCKOUT;
POLYPLOIDY;
SEX CHARACTERISTICS;
TRANSCRIPTION FACTORS;
TRANSPLANTATION, HOMOLOGOUS;
ANIMALIA;
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EID: 0242300080
PISSN: 00278424
EISSN: None
Source Type: Journal
DOI: 10.1073/pnas.2231861100 Document Type: Article |
Times cited : (80)
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References (21)
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