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Volumn 2, Issue 3, 2003, Pages 237-241
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Loss of mitotic spindle checkpoint activity predisposes to chromosomal instability at early stages of fibrosarcoma development.
a
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Author keywords
[No Author keywords available]
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Indexed keywords
CELL CYCLE PROTEIN;
CKS1 PROTEIN, S CEREVISIAE;
CYCLIN B;
PROTEIN P53;
SACCHAROMYCES CEREVISIAE PROTEIN;
ANIMAL;
ARTICLE;
BIOLOGICAL MODEL;
CELL CULTURE;
CELL TRANSFORMATION;
CHROMOSOME ABERRATION;
DISEASE COURSE;
DNA DAMAGE;
FIBROMA;
FIBROSARCOMA;
GENE;
GENETIC PREDISPOSITION;
GENETICS;
METABOLISM;
MITOSIS;
MITOSIS SPINDLE;
MOUSE;
MUTATION;
PHYSIOLOGY;
ANIMALS;
CELL CYCLE PROTEINS;
CELL TRANSFORMATION, NEOPLASTIC;
CHROMOSOME ABERRATIONS;
CYCLIN B;
DISEASE PROGRESSION;
DNA DAMAGE;
FIBROMA;
FIBROSARCOMA;
GENES, CDC;
GENETIC PREDISPOSITION TO DISEASE;
MICE;
MITOSIS;
MITOTIC SPINDLE APPARATUS;
MODELS, BIOLOGICAL;
MUTATION;
SACCHAROMYCES CEREVISIAE PROTEINS;
TUMOR CELLS, CULTURED;
TUMOR SUPPRESSOR PROTEIN P53;
MLCS;
MLOWN;
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EID: 0141834771
PISSN: 15384101
EISSN: None
Source Type: Journal
DOI: 10.4161/cc.2.3.355 Document Type: Article |
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Times cited : (9)
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References (0)
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