Mitochondrial dysfunction and reactive oxygen species in excitotoxicity and apoptosis: Implications for the pathogenesis of neurodegenerative diseases

Neurochemical Research

Volumn 28, Issue 10, 2003, Pages 1563-1574

  Rego, A Cristina ^a   Oliveira, Catarina R ^a  

^a UNIVERSITY OF COIMBRA   (Portugal)

Author keywords

Apoptosis; Calcium homeostasis; Excitotoxicity; Mitochondrial depolarization; Neurodegenerative diseases; Oxidative stress

Indexed keywords

ADENOSINE TRIPHOSPHATE; DNA FRAGMENT; MITOCHONDRIAL PROTEIN; REACTIVE OXYGEN METABOLITE; SUPEROXIDE;

APOPTOSIS; BRAIN MITOCHONDRION; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM TRANSPORT; CELL DEATH; CHROMATIN CONDENSATION; CYTOTOXICITY; DEGENERATIVE DISEASE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; EXCITOTOXICITY; NERVE DEGENERATION; OXIDATIVE PHOSPHORYLATION; OXIDATIVE STRESS; PATHOGENESIS; PRIORITY JOURNAL; PROTEIN AGGREGATION; REVIEW;

ALZHEIMER DISEASE; ANIMALS; APOPTOSIS; CALCIUM; HOMEOSTASIS; HUMANS; HUNTINGTON DISEASE; MITOCHONDRIA; NEURODEGENERATIVE DISEASES; PARKINSON DISEASE; REACTIVE OXYGEN SPECIES;

EID: 0141705360     PISSN: 03643190     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1025682611389     Document Type: Review

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