BRAF mutations and RET/PTC rearrangements are alternative events in the etiopathogenesis of PTC

Oncogene

Volumn 22, Issue 29, 2003, Pages 4578-4580

  Soares, Paula ^a,b   Trovisco, Vitor ^a   Rocha, Ana Sofia ^a   Lima, Jorge ^a   Castro, Patrícia ^a   Preto, Ana ^a   Máximo, Valdemar ^a   Botelho, Tiago ^a   Seruca, Raquel ^a   Sobrinho Simões, Manuel ^a,b,c  

^a UNIVERSITY OF PORTO   (Portugal)

^b Faculty of Engineering of Porto   (Portugal)

^c HOSPITAL DE SÃO JOÃO   (Portugal)

Author keywords

BRAF; Papillary carcinoma; RET PTC; Thyroid

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE;

ARTICLE; BRAF GENE; CANCER CELL CULTURE; CARCINOGENESIS; CELL STRAIN; CELL STRAIN TPC 1; CODON; ENZYME ACTIVATION; GENE; GENE ACTIVATION; GENE MUTATION; GENE REARRANGEMENT; GENETIC IDENTIFICATION; HUMAN; HUMAN CELL; MELANOMA; NODULAR GOITER; ONCOGENE RAS; ONCOGENE RET; PRIORITY JOURNAL; THYROID ADENOMA; THYROID FOLLICULAR CARCINOMA; THYROID PAPILLARY CARCINOMA;

CARCINOMA, PAPILLARY; DNA MUTATIONAL ANALYSIS; HUMANS; MUTATION; ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS B-RAF; PROTO-ONCOGENE PROTEINS C-RET; RAS PROTEINS; RECEPTOR PROTEIN-TYROSINE KINASES; THYROID NEOPLASMS; TUMOR CELLS, CULTURED;

EID: 0043170833     PISSN: 09509232     EISSN: None     Source Type: Journal    
DOI: 10.1038/sj.onc.1206706     Document Type: Article

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