The mechanisms of prednisone inhibition of inflammation in Crohn's disease involve changes in intestinal permeability, mucosal TNFα production and nuclear factor kappa B expression

Alimentary Pharmacology and Therapeutics

Volumn 18, Issue 3, 2003, Pages 309-317

  Wild, G E ^a,c   Waschke, K A ^a   Bitton, A ^a   Thomson, A B R ^b  

^a MCGILL UNIVERSITY   (Canada)

^b UNIVERSITY OF ALBERTA   (Canada)

^c MONTREAL GENERAL HOSPITAL   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; LACTULOSE; MANNITOL; PREDNISONE; TUMOR NECROSIS FACTOR ALPHA;

ADULT; ARTICLE; CLINICAL ARTICLE; COLON BIOPSY; CROHN DISEASE; DISEASE COURSE; FEMALE; HUMAN; INFLAMMATION; INTESTINE MUCOSA PERMEABILITY; MALE; PRIORITY JOURNAL; PROTEIN EXPRESSION; RELAPSE; REMISSION; TREATMENT OUTCOME;

ADULT; BLOTTING, WESTERN; CROHN DISEASE; FEMALE; HUMANS; I-KAPPA B PROTEINS; INTESTINAL MUCOSA; MALE; MIDDLE AGED; NF-KAPPA B; PERMEABILITY; PREDNISONE; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0041823880     PISSN: 02692813     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2036.2003.01611.x     Document Type: Article

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