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Volumn 17, Issue 11, 2003, Pages 1573-1575
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The function of GADD34 is a recovery from a shutoff of protein synthesis induced by ER stress: elucidation by GADD34-deficient mice.
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Author keywords
[No Author keywords available]
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Indexed keywords
CARRIER PROTEIN;
CCAAT ENHANCER BINDING PROTEIN;
CELL CYCLE PROTEIN;
CHAPERONE;
DDIT3 PROTEIN, MOUSE;
DIFFERENTIATION ANTIGEN;
DITHIOTHREITOL;
GROWTH ARREST AND DNA DAMAGE INDUCIBLE PROTEIN 153;
HEAT SHOCK PROTEIN;
INITIATION FACTOR 2;
MOLECULAR CHAPERONE GRP78;
PPP1R15A PROTEIN, MOUSE;
PROTEIN;
THAPSIGARGIN;
TRANSCRIPTION FACTOR;
TUNICAMYCIN;
ANIMAL;
ARTICLE;
BIOLOGICAL MODEL;
C57BL MOUSE;
DRUG EFFECT;
ENDOPLASMIC RETICULUM;
FETUS;
GENETICS;
METABOLISM;
MOUSE;
MOUSE MUTANT;
PHOSPHORYLATION;
PHYSIOLOGY;
PRENATAL DEVELOPMENT;
PROTEIN SYNTHESIS;
TISSUE DISTRIBUTION;
ANIMALS;
ANTIGENS, DIFFERENTIATION;
CARRIER PROTEINS;
CCAAT-ENHANCER-BINDING PROTEINS;
CELL CYCLE PROTEINS;
DITHIOTHREITOL;
EMBRYONIC AND FETAL DEVELOPMENT;
ENDOPLASMIC RETICULUM;
EUKARYOTIC INITIATION FACTOR-2;
FETUS;
HEAT-SHOCK PROTEINS;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
MODELS, BIOLOGICAL;
MOLECULAR CHAPERONES;
PHOSPHORYLATION;
PROTEIN BIOSYNTHESIS;
PROTEINS;
THAPSIGARGIN;
TISSUE DISTRIBUTION;
TRANSCRIPTION FACTOR CHOP;
TRANSCRIPTION FACTORS;
TUNICAMYCIN;
MLCS;
MLOWN;
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EID: 0041703031
PISSN: None
EISSN: 15306860
Source Type: Journal
DOI: 10.1096/fj.02-1184fje Document Type: Article |
Times cited : (198)
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References (0)
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